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I mars 2015 utvärderade 17 experter från 11 länder vid WHO’s cancerorgan IARC cancerrisker av bekämpningsmedel däribland glyfosat. Det är ett storsäljande ogräsmedel under bland annat namnet Roundup, som används i jordbruket men även privat i villaträdgårdar etc. Det finns att köpa även i sprayflaskor för besprutning av grusgångar, garageuppfarter osv. Glyfosat kan återfinnas t.ex. i blod, bröstmjölk och urin främst hos jordbruksarbetare. Även förgiftning av vatten har påvisats i vissa områden.

Utvärderingen grundades både på studier av människa och djur. Glyfosat leder till skador på DNA och kromosomer. Ökad risk har visats bland besprutare för lymfom (non-Hodgkin lymfom), leukemier och även en del andra cancerformer. Ökad cancerrisk har även visats i djurstudier. Våra studier har visat ökad risk för non-Hodgkin lymfom, vilka ingick i beslutsunderlaget.

Experterna vid IARC bedömde glyfosat som ”troligen” cancerframkallande för människa (probably carcinogenic to humans Group 2A).

Monsanto är ett av de företag som tillverkar glyfosat. Naturligtvis har beslutet vid IARC lett till kraftig motreaktion från industrin. Det är märkligt att det i Sverige saknas debatt om preparatet som finns fritt tillgängligt för allmänheten. Självklart borde glyfosat dras in från marknaden och om något endast få användas av personer med licens för besprutning, något som gjorts i vissa länder.

Viss debatt finns internationellt, se t.ex.

http://www.theguardian.com/cities/2015/apr/21/glyphosate-probably-carcinogenic-pesticide-why-cities-use-it

Eller

http://www.independent.co.uk/news/science/glyphosate-scientists-urge-caution-over-experts-claims-pesticide-is-probably-carcinogenic-10397787.html

Non-Hodgkin lymfom orsakas av kemikalier som påverkar immunsystemet och ger andra skador. Exempel på sådana kemikalier är PCB, dioxiner och bekämpningsmedel. Sedan 1970 har det skett en kraftig ökning av antalet personer som insjuknar enligt cancerregistret. Denna trend får ses som ett uttryck för den ökning av cancerframkallande ämnen, t.ex. miljögifter, som vi utsätts för.

ICD-7 - 200

Det första utkastet till monografi om hälsoeffekter av radiofrekventa fält från WHO innehåller många sakfel som vi påpekat tidigare. Vi har nu påtalat detta på nytt och skickat till WHO brev enligt nedan. IARC har fått samma brev med begäran om ny utvärdering av cancerrisker.

 

 

World Health Organization                                                              4 August, 2015

 

Dr Margaret Chan, Director General

World Health Organization

Avenue Appia 20, 1211 Geneva 27

Geneva, Switzerland

 

Emelie van Deventer, Team Leader

Radiation Programme Department of Public Health,

Environmental and Social Determinants of Health,

World Health Organization

Geneva, Switzerland

 

Dear Ms. Margaret Chan

Dear Ms. Emelie van Deventer

 

Further Comments on the WHO draft: Radio Frequency fields: Environmental Health Criteria Monograph

On 15 December, 2014 we submitted comments on the WHO draft on radio frequency fields and health. Since we have not got a satisfactory reply from WHO, not seen a revision of the draft, and adding to that more published studies that reinforce the increased risk for certain brain tumours associated with use of wireless phones we want to submit the following, additional comments.

The brain is the primary target organ for exposure to radiofrequency electromagnetic fields (RF-EMF) during the use of the handheld wireless phone. This has given concern of an increased risk for brain tumours. The carcinogenic effect of RF-EMF on humans was evaluated at a meeting during 24 – 31 May 2011 at the International Agency for Research on Cancer (IARC) at WHO in Lyon, France. One of us (LH) was part of the expert group. The Working Group categorised RF-EMF from mobile phones, and from other devices that emit similar non-ionising electromagnetic fields in the frequency range 30 kHz–300 GHz, as a Group 2B, i.e. a possible, human carcinogen (http://monographs.iarc.fr/ENG/Monographs/vol102/mono102.pdf).

Since then more studies have been published that strengthen the association between use of wireless phones (mobile and cordless phones) and increased risk for brain tumours. We have performed long-term research in this area and in the following we give a short up-dated summary of our findings based on research since the 1990’s. In our publications relevant information can be found also on other studies, as well as discussions of the current scientific evidence.

Glioma:

Glioma is a malignant brain tumour (“brain cancer”), and the most common type is glioblastoma multiforme with a poor prognosis. We have published a statistically significant increased risk for glioma among users of both mobile and cordless phones. The risk increased with latency (time from first use of the phone until tumour diagnosis) and cumulative number of hours for use. Highest risk was found in the area of the brain with highest exposure to RF-EMF. All these results are of biological relevance; that is what would be expected for a causal association. The full paper can be read here:

http://www.pathophysiologyjournal.com/article/S0928-4680(14)00064-9/pdf

Meningioma:

Menigioma is mostly a benign brain tumour and accounts for about 30 % of all intracranial tumours. The incidence is approximately 2-times higher in women than in men. No conclusive evidence of an association between use of mobile and cordless phones and meningioma was found in our study. However, taking the long latency periods that have been reported for the increased meningioma risk associated with exposure to ionizing radiation it is still too early to make a definitive risk assessment. Results for even longer latency periods of wireless phone use than in our study are desirable, see more details here:

http://www.spandidos-publications.com/or/33/6/3093

Acoustic neuroma:

Acoustic neuroma or Vestibular Schwannoma is a rare benign tumour in the eighth cranial nerve that leads from the inner ear to the brain. It grows slowly and does not undergo malignant transformation, but may give compression of vital brain stem centres. Tinnitus and hearing problems are usual first symptoms of acoustic neuroma. We published a clear, statistically significant, association between use of mobile and cordless phones and acoustic neuroma. The risk increased with time since first use. For use of both mobile and cordless phones the risk was highest in the longest latency group. Tumour volume increased per 100 hours of cumulative use and year of latency for wireless phones indicating tumour progression from RF-EMF. The whole study can be read here:

http://www.spandidos-publications.com/ijo/43/4/1036

Brain tumour prognosis:

A causal association would be strengthened if use of wireless phones has an impact on the survival of glioma patients. We analyzed survival of 1,678 glioma patients in our case-control studies 1997-2003 and 2007-2009. Use of wireless phones in the > 20 years latency group (time since first use) yielded increased hazard ratio (HR) = 1.7, 95 % confidence interval (CI) = 1.2-2.3 for glioma, i.e. decreased survival. Increased HR was found for use of both mobile and cordless phones. Highest HR was found for cases with first use before the age of 20 years. These results strengthen a causal association between use of wireless phones and glioma. The publication can be read here:

http://www.mdpi.com/1660-4601/11/10/10790

Risk in different age groups of first use:

In our glioma study we found highest risk for subjects with first use of mobile or cordless phone before the age of 20, see Table 8 in the publication:

http://www.pathophysiologyjournal.com/article/S0928-4680(14)00064-9/pdf

We published similar results for acoustic neuroma and use of mobile phones, see Table 21.2:

http://citeseerx.ist.psu.edu/viewdoc/download?doi=10.1.1.433.7480&rep=rep1&type=pdf

Children and adolescents are more exposed to RF-EMF than adults due to thinner skull bone, higher conductivity in the brain tissue, and a smaller head. The developing brain is also more vulnerable than in adults and it is still developing until about 20 years of age. The finding of higher risk in young persons is worrying, not the least due to the high prevalence of use of wireless phones in children and adolescents.

Brain tumour incidence:

It is not correct to claim that the incidence of brain tumours has not increased in the Scandinavian countries. The age-standardized incidence of brain tumours increased dramatically in Denmark with +41.2 % among men and +46.1 % among women during 2003-2012 (http://www.ssi.dk/Aktuelt/Nyheder/2013/~/media/Indhold/DK – dansk/Sundhedsdata og it/NSF/Registre/Cancerregisteret/Cancerregisteret 2012.ashx).

Due to the well-known under-reporting of brain tumours to the Swedish Cancer Registry we studied brain tumour rates using the Swedish National Inpatient Register and the Causes of Death Register (see http://www.mdpi.com/1660-4601/12/4/3793/htm ). In summary we found a statistically significant increasing rate of not specified brain tumours from 2007 in the Inpatient Register and from 2008 in the Causes of Death Register. Our study indicated that several of these tumours were never reported to the Swedish Cancer Register. The results are in accordance with a reasonable latency period for use of wireless phones, e.g. mobile phones, see Figures 5 and 6 in our publication. Thus, the Swedish Cancer Register data cannot be used to dismiss an increased risk for brain tumours associated with use of wireless phones. On the contrary our study is consistent with an association considering a reasonable tumour induction period.

Mechanistic aspects:

Reactive oxygen species:

RF-EMFs do not cause direct DNA damage. On the other hand numerous studies have shown generation of reactive oxygen species (ROS) that can cause oxidative damage of DNA. This is a well-known mechanism in carcinogenesis for many agents. The broad biological potential of ROS and other free radicals makes radiofrequency radiation a potentially hazardous factor for human health, not only cancer risk but also other health effects. A recent update can be read here:

http://informahealthcare.com/doi/abs/10.3109/15368378.2015.1043557

-Tumour promotion:

Tumour promotion by RF-EMF exposure was reported in 2010 in a study on mice: http://www.ncbi.nlm.nih.gov/pubmed/20545575. These findings were recently replicated and add to the relevance of tumour risk: http://www.ncbi.nlm.nih.gov/pubmed/25749340

-p53:

The p53 protein is a transcription factor that plays a vital role in regulating cell growth, DNA repair and apoptosis, and p53 mutations are involved in disease progression. In a recent study it was found that use of mobile phones for ≥3 hours a day was associated with increased risk for the mutant type of p53 gene expression in the peripheral zone of astrocytoma grade IV (glioblastoma multiforme), and that this increase was statistically significant correlated with shorter overall survival time:

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4178273/

 These results are in agreement with the decreased survival for patients with astrocytoma grade IV (glioblastoma multiforme) associated with long-term use of mobile phones and cordless phones that we reported in 2014, see above the section on prognosis.

Causality:

To further evaluate strengths of evidence Sir Austin Bradford Hill wrote in the 1960’s a famous article on association or causation at the height of the tobacco and lung cancer controversy: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1898525/pdf/procrsmed00196-0010.pdf

Hill offered a list of nine aspects of an association to be considered when deciding if an association is causal. However, he did not request all nine viewpoints to be fulfilled for causality. We used the Hill criteria to evaluate the causality on brain tumour risk from RF-EMF emitted from wireless phones. We concluded that based on the Hill criteria, glioma and acoustic neuroma should be considered to be caused by RF-EMF emissions from wireless phones and regarded as carcinogenic to humans, classifying it as Group 1 according to the IARC classification. See more here:

http://www.degruyter.com/view/j/reveh.2013.28.issue-2-3/reveh-2013-0006/reveh-2013-0006.xml

Conclusion:

Our results are in agreement with other studies such as the international Interphone study and the French CERENAT study. This is discussed in more detail in e.g. our article on glioma risk, see also:

http://www.pathophysiologyjournal.com/article/S0928-4680(12)00110-1/pdf

The so called Danish cohort study on mobile phone users has been taken as evidence of no risk. However, the many shortcomings as reviewed elsewhere makes the study inconclusive regarding assessment of cancer risk. It should not be cited as evidence of no risk, for more details see: http://www.degruyter.com/view/j/reveh.2012.27.issue-1/reveh-2012-0004/reveh-2012-0004.xml?format=INT

In summary there is consistent evidence of increased risk for glioma and acoustic neuroma associated with use of mobile phones and cordless phones. Furthermore, the risk is highest for persons with first use before the age of 20, which is of special concern. Our conclusion is that RF-EMF should be regarded as a human carcinogen. The IARC classification should be updated to at least Group 2A, a probable human carcinogen. Current guidelines for exposure need to be urgently revised. The WHO Monograph draft on this issue is based on selective inclusion of studies and wrong assessment of the evidence of increased risk. Thus the Danish cohort study on mobile phone users and the Swedish Cancer Register data cannot be used as evidence of no increased risk. It is important that the public and decision makers are given correct information about the cancer risk so that they can make decisions based on correct data and take precautions. Otherwise there is an obvious risk of forthcoming increasing impairment of human health and increasing numbers of cancer in the population. We anticipate correction of the Monograph and your reply to this letter no later than 15 September, 2015. If you so wish our research group may of course give a presentation at WHO on this topic.

Yours sincerely,

 

Lennart Hardell, MD, PhD

Department of Oncology

University Hospital

SE-701 85 Örebro

Sweden

 

Michael Carlberg, MSc

Department of Oncology

University Hospital

SE-701 85 Örebro

Sweden

Ms. Margaret Sullivan, Public Editor                                       July 24, 2015

Ms. Carol Pogas, Reporter

 

The New York Times

Regarding: Cellphone Ordinance Puts Berkeley at Forefront of Radiation Debate http://www.nytimes.com/2015/07/22/us/cellphone-ordinance-puts-berkeley-at-forefront-of-radiation-debate.html?_r=0

Published online July 21, 2015

Dear Ms. Sullivan and Ms. Pogas,

We have read this article in the New York Times with interest. However, there are several mistakes, and even wrong statements, on the health hazards from exposure to radiofrequency electromagnetic fields (RF-EMF) from cell phones in the article. In the following we want to correct some of the false statements.

The brain is the primary target organ for exposure to RF-EMF during the use of the handheld wireless phone. This has given concern of an increased risk for brain tumours. The carcinogenic effect of RF-EMF on humans was evaluated at a meeting during 24 – 31 May 2011 at the International Agency for Research on Cancer (IARC) at WHO in Lyon, France. One of us (LH) was part of the expert group. The Working Group categorised RF-EMF from mobile phones, and from other devices that emit similar non-ionising electromagnetic fields in the frequency range 30 kHz–300 GHz, as a Group 2B, i.e. a possible, human carcinogen (http://monographs.iarc.fr/ENG/Monographs/vol102/mono102.pdf).

Since then more studies have been published that strengthen the association between use of  wireless phones (mobile and cordless phones) and increased risk for brain tumours. We have performed long-term research in this area and in the following we give a short up-dated summary of our findings based on research since the 1990’s. In our publications relevant information can be found also on other studies, as well as discussions of the current scientific evidence.

Glioma:

Glioma is a malignant brain tumour (“brain cancer”), and the most common type is glioblastoma multiforme with a poor prognosis. We have published a statistically significant increased risk for glioma among users of both mobile and cordless phones. The risk increased with latency (time from first use of the phone) and cumulative number of hours for use. Highest risk was found in the area of the brain with highest exposure to RF-EMF. All these results are of biological relevance; that is what would be expected for a causal association. The full paper can be read here: http://www.pathophysiologyjournal.com/article/S0928-4680(14)00064-9/pdf

Meningioma:

Meningioma is mostly a benign brain tumour and accounts for about 30 % of all intracranial tumours. The incidence is approximately 2-times higher in women than in men. No conclusive evidence of an association between use of mobile and cordless phones and meningioma was found in our study. However, taking the long latency periods that have been reported for the increased meningioma risk associated with exposure to ionizing radiation it is still too early to make a definitive risk assessment. Results for even longer latency periods of wireless phone use than in our study are desirable, see more details here: http://www.spandidos-publications.com/or/33/6/3093

 Acoustic neuroma:

Acoustic neuroma or Vestibular Schwannoma is a rare benign tumour in the eighth cranial nerve that leads from the inner ear to the brain. It grows slowly and does not undergo malignant transformation, but may give compression of vital brain stem centres. Tinnitus and hearing problems are usual first symptoms of acoustic neuroma. We published a clear, statistically significant, association between use of mobile and cordless phones and acoustic neuroma. The risk increased with time since first use. For use of both mobile and cordless phones the risk was highest in the longest latency group. Tumour volume increased per 100 hours of cumulative use and year of latency for wireless phones indicating tumour progression from RF-EMF. The whole study can be read here: http://www.spandidos-publications.com/ijo/43/4/1036

Brain tumour incidence:

It is not correct to claim that the incidence of brain tumours has not increased in the Scandinavian countries. The age-standardized incidence of brain tumours increased dramatically in Denmark with +41.2 % among men and +46.1 % among women during 2003-2012 (http://www.ssi.dk/Aktuelt/Nyheder/2013/~/media/Indhold/DK – dansk/Sundhedsdata og it/NSF/Registre/Cancerregisteret/Cancerregisteret 2012.ashx).

Due to the well-known under-reporting of brain tumours to the Swedish Cancer Registry we studied brain tumour rates using the Swedish National Inpatient Register and the Causes of Death Register. In summary we found a statistically significant increasing rate of not specified brain tumours from 2007 in the Inpatient Register and from 2008 in the Causes of Death Register. Our study indicated that several of these tumours were never reported to the Swedish Cancer Register. Thus, the Swedish Cancer Register data cannot be used to dismiss an increased risk for brain tumours associated with use of wireless phones. On the contrary our study is consistent with an association considering a reasonable tumour induction period, see more here: http://www.mdpi.com/1660-4601/12/4/3793

Mechanistic aspects:

It is correct that RF-EMFs do not cause direct DNA damage. On the other hand numerous studies have shown generation of reactive oxygen species (ROS) that can cause oxidative damage of DNA. This is a well-known mechanism in carcinogenesis for many agents. The broad biological potential of ROS and other free radicals makes radiofrequency radiation a potentially hazardous factor for human health, not only cancer risk but also other health effects. A recent update can be read here: http://informahealthcare.com/doi/abs/10.3109/15368378.2015.1043557

Causality:

To further evaluate strengths of evidence Bradford Hill wrote in the 1960’s a famous article on association or causation at the height of the tobacco and lung cancer controversy. Hill offered a list of nine aspects of an association to be considered when deciding if an association is causal. However, he did not request all nine viewpoints to be fulfilled for causality. We used the Hill criteria to evaluate the causality on brain tumor risk from RF-EMF emitted from wireless phones. We concluded that based on the Hill criteria, glioma and acoustic neuroma should be considered to be caused by RF-EMF emissions from wireless phones and regarded as carcinogenic to humans, classifying it as Group 1 according to the IARC classification. Current guidelines for exposure need to be urgently revised. See more here: http://www.degruyter.com/view/j/reveh.2013.28.issue-2-3/reveh-2013-0006/reveh-2013-0006.xml

Conclusion:

Our results are in agreement with other studies such as the international Interphone study and the French so called CERENAT study. This is discussed in e.g. our article on glioma risk. In summary there is consistent evidence of increased risk for glioma and acoustic neuroma associated with use of mobile phones and cordless phones. Furthermore, the risk is highest for persons with first use before the age of 20, which is of special concern. Our conclusion is that RF-EMF should be regarded as a human carcinogen. The IARC classification should be updated to at least Group 2A, a probable human carcinogen. It is necessary to give the public correct information on the cancer risk. The precautionary principle should be used to minimize exposure to RF-EMF. Media have an important role to inform in a balanced way. Unfortunately this article in the New York Times is biased towards the no risk assumption. It should be corrected based on facts and not wishful thinking.

Yours sincerely,

 

Lennart Hardell, MD, PhD

Department of Oncology

University Hospital

SE-701 85 Örebro

Sweden

 

Michael Carlberg, MSc

Department of Oncology

University Hospital

SE-701 85 Örebro

Sweden

 

 

I en uppföljande artikel i NewsVoice med titeln Hans-Olov Adami – Svensk mästare i att avfärda cancerrisker: ”Tjänar inte samhället att cancerklassa dioxin” diskuterar vetenskapsjournalisten Mona Nilsson en kontroversiell professor vid KI och Harvard med stora forskningsbidrag från Cancerfonden för att minska risken för cancer. Resultatet tycks ha blivit det motsatta. Här tycks stora bidrag från industrin för att tvätta bort cancerrisker (greenwashing) ha spelat in. Vilka etiska, moraliska och vetenskapliga principer råder (eller snarast saknas) vid Cancerfonden? Detta synes vara misshushållning av pengar insamlade från allmänheten. Artikeln är läsvärd och ingressen lyder:

”NewsVoice berättade i tidigare artikel om Cancerfondens fortsatta miljonstöd till den kontroversielle professorn Hans-Olov Adami, trots att det var känt sedan 2001 att Adami parallellt fick betalt av kemiindustrin och konsultbolaget Exponent för att avfärda cancerrisker med cancerklassade kemikalier. Hans Olov Adami fick under perioden 2002 -2014 22 miljoner kronor av Cancerfonden. Under de senaste åren har han avfärdat cancerrisker med Agent Orange, bekämpningsmedel, syntetiska kemikalier och formaldehyd.”

I ett brev daterat den 12 mars 2015 skriver Stefan Bergh vid Cancerfonden att det inte finns något samband mellan mobiler och hjärntumörer: ”Tala i mobil ökar inte risk för hjärntumör” se nedan.

Uttalandet stämmer inte överens med utvärderingen vid WHO-organet IARC i maj 2011 varvid radiofrekventa fält t.ex. från trådlösa telefoner klassades som ’möjligen cancerframkallande’ för människa, Grupp 2B. Sambandet har därefter stärkts i fortsatta studier.

Fler och fler personer i Sverige får cancer. Det är då viktigt att Cancerfonden ger korrekt information för att kunna minska cancerriskerna.  Vår forskning om mobiler och risken för hjärntumörer har bekostats främst av Cancer- och Allergifonden och Cancerhjälpen i Sverige, däremot inte av Cancerfonden.

 

cancerfonden

Vi publicerade nyligen resultat av våra epidemiologiska studier av risken för hjärntumör av typen meningiom och användning av trådlösa telefoner (mobiltelefoner och trådlösa bordstelefoner; DECT). Studieperioderna omfattade svenska patienter som diagnostiserats 1997-2003 eller 2007-2009; totalt 1625 personer deltog. Jämförelsegruppen bestod av 3500 personer ur befolkningsregistret.

Sammantaget sågs ingen tydligt ökad risk även om risken var något förhöjd för personer med störst antal timmar för användning av trådlös telefon och längst latenstid (antal år från första användning till diagnos av meningiom). Resultaten stämmer överens med andra undersökningar inom området och skiljer sig från våra resultat för gliom (’hjärncancer’) och acusticusneurinom (tumör på hörselnerven) där vi sett ett klart samband med trådlösa telefoner.

Studien är publicerad i en vetenskaplig tidning efter sedvanlig förhandsgranskning och kan återfinnas här.

En av Cancerfondens viktigaste uppgifter är att minska risken för cancer så att färre drabbas. Utvecklingen är den motsatta även om hänsyn tas till åldersstrukturen i befolkningen. År 2013 diagnosticerades över 61 000 nya cancerfall – sannolikt har fler drabbats eftersom det numera finns stora brister i rapporteringen till cancerregistret. Siffran kan jämföras med exempelvis 1970 med drygt 28 000 nya cancerfall. Ökningen är anmärkningsvärd även om hänsyn tas till att vi blir fler och äldre. Cancer är numera en folksjukdom!

Att identifiera cancerrisker och verka för åtgärder genom information och lagstiftning för att minska sådan exponering i befolkningen borde vara en av huvuduppgifterna för Cancerfonden. Exempel på risker där Cancerfonden varit passiv är bekämpningsmedel, dioxiner, PCB och nu senast mobiltelefoner och hjärntumörer. Informationen har under många år ensidigt gällt personliga riskfaktorer som kost, motion, övervikt och rökning.

Det har under lång tid varit känt att en svensk cancerforskare med personlig forskartjänst från Cancerfonden syftande till att identifiera och minska cancerrisker, och även med stora forskningsanslag, arbetat för industrin med avsikt att bortförklara cancerrisker. Trots att Cancerfonden känt till detta har förhållandet uppenbarligen inte haft betydelse för fortsatta stora penningmängder från Cancerfonden till denne forskare.

NewsVoice publicerade den 3 juni 2015 en artikel om denna skandal som svärtar ner Cancerfonden. Trots allt gör majoriteten av de forskare som Cancerfonden stöttar bra forskning, inte minst för att förstå de biologiska mekanismerna för cancer och att få fram bättre behandlingar. En persons övertramp får givetvis inte skymma Cancerfondens övriga uppgifter. Onekligen är dock prevention det bästa vapnet mot cancer varför Cancerfondens bidrag till forskare som bortförklarar cancerrisker är svårbegriplig.

 Ingressen till artikeln lyder:

”Cancerfonden har under 2 decennier betalat sammanlagt över 30 miljoner till den kontroversiella professorn Hans-Olov Adami. Adami kritiserades redan för över tio år sedan för sitt nära samarbete med kemikalieindustrin. Trots det har Cancerfonden fortsatt att betala ut över 20 miljoner till Professor Adami. Under samma tid har Adami avfärdat cancerrisker med alltifrån Agent Orange, dioxin, formaldehyd, bekämpningsmedel och mobilstrålning. Antalet människor som får cancer i Sverige har ökat med 30% under samma period.”